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Six
million people a year in Britain are affected by ulcers. Although they
become more common with age, they are, according to the Digestive
Disorders Foundation, becoming increasingly prevalent among younger
people.
Until recently, it was generally
thought that ulcers were caused by stress, spicy food and/or excess
stomach acid. While this may be somewhat true, it is now thought that up
to 90 per cent of peptic ulcers result from a stomach infection caused by
the bacteria Helicobacter pylori (H. pylori).
Thus, the new conventional treatment strategy for gastric ulcers is now
antibiotic therapy rather than anti-acids, or both together.
It has been found that at least 90% of those with duodenal ulcers, 70%
with gastric ulcers, and 50% of people over the age of 50 test positive
for the presence of H.pylori. Indeed, about 30% of all people in the UK
are probably infected with the bacteria. However, many of those who test
positive do not develop ulcers and remain symptom-free. An evaluation of
the presence of the bacteria can be performed using blood, saliva, stool
or a breath test.
This bacterium lives in the stomach of
almost half of the world's population. It gets into the stomach and stays
there until eradicated. The World Health Organisation now considers this
bacterium a carcinogen (cancer producing).
Why H. pylori does not cause ulcers in every infected person is not
known. Most likely, infection depends on characteristics of the infected
person, the type of H. pylori, and other factors yet to be discovered.
Researchers are not certain how people contract H. pylori, but they
think it may be through food or water. It has also been found in some
infected people’s saliva, so mouth-to-mouth contact, such as kissing, may
spread the bacteria. Faecal to oral route and houseflies landing on food
are also possible causes of infection.
The formation of ulcers occur when the lining of the stomach and small
intestine are thinned or damaged. The intestinal lining has a protective
coating of mucosa, which protects the stomach and duodenum from becoming
damaged by gastric acid. Unlike the H.pylori bacteria, most pathogenic
micro-organisms cannot survive the acidic environment of the stomach.
However, the bacterium H. pylori first neutralises the gastric pH in the
immediate area, then "drills" into the unprotected mucosal lining, causing
gastritis, which in turn can lead to the formation of ulcers.
What are the symptoms?
The symptoms of a peptic ulcer include chronic burning or gnawing
stomach pain that usually begins forty-five to sixty minutes after eating
or at night, and that is relieved by eating, taking antacids, vomiting, or
drinking a large glass of water. The pain may range from mild to severe.
It may cause the sufferer to awaken in the middle of the night. Other
symptoms include excess belching, loss of appetite, nausea, lower back
pain and headaches.
Complications such as bleeding from the ulcer itself can be very
dangerous. If symptoms include vomiting blood, blood in the faeces or
black faeces, seek emergency medical assistance.
Sometimes the ulcer can perforate the stomach, causing an actual hole
in the stomach wall that allows bacteria from the bowel to enter it. This
can cause peritonitis – which unless effectively treated is rapidly fatal.
Symptoms of a perforated ulcer include sudden severe abdominal pain
followed by pain spreading to the back. Always consult your medical
practitioner if in doubt.
It is often necessary to perform tests to distinguish between gastric
ulcers, duodenal ulcers and stomach cancer.
In a barium test, a salt that is insoluble in water and appears opaque
on the X-rays, is swallowed. The X-ray shows up the salt as it travels
through the digestive system, revealing a clear outline of the stomach and
duodenum.
If this suggests an ulcer, an endoscopy (where a fibre-optic cable is
passed through the mouth and into the stomach) and biopsy are usually
performed to make certain of the diagnosis.
What are the treatments?
Conventional treatments are usually antibiotics and/or antacids.
Mastic Gum: A Breakthrough
alternative treatment
Mastic gum, a resin produced by the
Pistacia lentiscus tree (an evergreen shrub from the pistachio tree
family), has been used for a variety of gastric ailments in Mediterranean
and Middle Eastern countries for at least 3,000 years. In ancient times,
mastic gum was highly revered for its medicinal properties in the relief
of dyspepsia and other intestinal disorders. The benefits of this
naturally-occurring resin is now being rediscovered for its antimicrobial
effects.
(please
read this important notice concerning supplement medical claims)
Studies show Mastic Gum kills H.
Pylori
Several studies have already been published on mastic gum with regard
to its positive effects on the gastrointestinal environment, thus gaining
respect among the scientific and medical community. Perhaps the most
exciting breakthrough to date is that of a recent study showing mastic
gum’s effectiveness against at least seven different strains of
Helicobacter pylori.
In other studies, mastic gum provided symptomatic relief of ulcers,
reduced the intensity of gastric mucosal damage caused by anti-ulcer drugs
and aspirin, and possessed antacid and cytoprotective qualities. In
several studies using mastic gum on patients with ulcers, the original
site of the ulcer was completely replaced by healthy epithelial cells.
Other benefits of Mastic Gum
In Middle Eastern and Mediterranean
countries mastic gum has been used as a food and confectionary additive
for a long time. It has been used to prevent dental carries, heal mouth
ulcers and other gum problems, control diabetes, reduce cholesterol levels
and treat skin ulcers. In some parts of Africa, Mastic has been used as an
aphrodisiac to boost sexual performance.
After centuries of extensive use in
Mediterranean and Middle Eastern countries, both as a dietary supplement
and herbal remedy, no undesirable effects have ever been attributed to
mastic. The medical trials also showed mastic to have no side effects.
Clinical experience
Leo Galland MD of New York City is well
known for his expertise with chronic gastric disorders, intestinal
permeability ("leaky gut syndrome"), ulcers, and dyspepsia. This work has
led him to use mastic gum with his patients, especially when H. pylori is
present. Says Dr Galland on mastic gum, "I am treating patients with
dyspepsia and gastritis who also have Helicobacter in the stool. I do not
use synthetic antibiotics, just mastic gum. In my experience, a dosage of
500mg to 1 gram twice a day for two weeks has produced a clearing of
symptoms associated with the elimination of the Helicobacter antigen from
the stool. Ninety percent experience a clearing of symptoms, and 80%
experience an elimination of Helicobacter in the stool after only two
weeks.
Please note this article does not replace the
advice and treatment of your medical practitioner.
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